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Abstract
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The research investigating the relationship between HPV E7 gene expression and Snail1/Snail2 gene expression in cervical cancer is crucial for several reasons. Cervical cancer, primarily caused by high-risk HPV types, poses significant public health challenges, particularly in developing regions. Understanding how the E7 oncogene promotes tumorigenesis and interacts with Snail transcription factors could reveal mechanisms of metastasis and identify new therapeutic targets. This study may lead to novel strategies for disrupting the interactions between E7 and Snail proteins, enhancing treatment efficacy. Additionally, it could contribute to the development of biomarkers for disease progression and inform therapeutic vaccine strategies for patients already infected with HPV. The research also addresses health disparities by improving early detection and treatment in underserved populations. Furthermore, it encourages interdisciplinary collaboration across fields such as virology, oncology, and public health. Ultimately, this investigation has the potential to enhance our understanding of cervical cancer biology, improve prevention and treatment approaches, and raise awareness about the importance of HPV vaccination and regular screening.
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